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The role of the mitochondrial protein VDAC1 in inflammatory bowel disease:  a potential therapeutic target: Molecular Therapy
The role of the mitochondrial protein VDAC1 in inflammatory bowel disease: a potential therapeutic target: Molecular Therapy

Reduction of inflammation and colon injury by a Pennyroyal phenolic extract  in experimental inflammatory bowel disease in mice - ScienceDirect
Reduction of inflammation and colon injury by a Pennyroyal phenolic extract in experimental inflammatory bowel disease in mice - ScienceDirect

Immuno-PET of Innate Immune Markers CD11b and IL-1β Detects Inflammation in  Murine Colitis | Journal of Nuclear Medicine
Immuno-PET of Innate Immune Markers CD11b and IL-1β Detects Inflammation in Murine Colitis | Journal of Nuclear Medicine

IJMS | Free Full-Text | Dysbiosis in Inflammatory Bowel Disease: Pathogenic  Role and Potential Therapeutic Targets | HTML
IJMS | Free Full-Text | Dysbiosis in Inflammatory Bowel Disease: Pathogenic Role and Potential Therapeutic Targets | HTML

An Integrated Taxonomy for Monogenic Inflammatory Bowel Disease -  Gastroenterology
An Integrated Taxonomy for Monogenic Inflammatory Bowel Disease - Gastroenterology

Cells | Free Full-Text | Roles of Autophagy-Related Genes in the  Pathogenesis of Inflammatory Bowel Disease | HTML
Cells | Free Full-Text | Roles of Autophagy-Related Genes in the Pathogenesis of Inflammatory Bowel Disease | HTML

DSS-induced colitis is associated with adipose tissue dysfunction and  disrupted hepatic lipid metabolism leading to hepatosteatosis and  dyslipidemia in mice | Scientific Reports
DSS-induced colitis is associated with adipose tissue dysfunction and disrupted hepatic lipid metabolism leading to hepatosteatosis and dyslipidemia in mice | Scientific Reports

Antibody signatures in inflammatory bowel disease: current developments and  future applications: Trends in Molecular Medicine
Antibody signatures in inflammatory bowel disease: current developments and future applications: Trends in Molecular Medicine

Molecular classification of Crohn's disease reveals two clinically relevant  subtypes | Gut
Molecular classification of Crohn's disease reveals two clinically relevant subtypes | Gut

IJMS | Free Full-Text | Inflammatory Bowel Disease: New Insights into the  Interplay between Environmental Factors and PPARγ | HTML
IJMS | Free Full-Text | Inflammatory Bowel Disease: New Insights into the Interplay between Environmental Factors and PPARγ | HTML

Recent advances in inflammatory bowel disease: mucosal immune cells in intestinal  inflammation | Gut
Recent advances in inflammatory bowel disease: mucosal immune cells in intestinal inflammation | Gut

Evaluation of anti-TNF therapeutic response in patients with inflammatory  bowel disease: Current and novel biomarkers - eBioMedicine
Evaluation of anti-TNF therapeutic response in patients with inflammatory bowel disease: Current and novel biomarkers - eBioMedicine

Cytokines in inflammatory bowel disease | Nature Reviews Immunology
Cytokines in inflammatory bowel disease | Nature Reviews Immunology

IJMS | Free Full-Text | Inflammatory Bowel Disease Treatments and  Predictive Biomarkers of Therapeutic Response | HTML
IJMS | Free Full-Text | Inflammatory Bowel Disease Treatments and Predictive Biomarkers of Therapeutic Response | HTML

Uncovering the cause of ulcerative colitis - Roberts‐Thomson - 2019 - JGH  Open - Wiley Online Library
Uncovering the cause of ulcerative colitis - Roberts‐Thomson - 2019 - JGH Open - Wiley Online Library

Targeting uPA-uPAR interaction to improve intestinal epithelial barrier  integrity in inflammatory bowel disease - eBioMedicine
Targeting uPA-uPAR interaction to improve intestinal epithelial barrier integrity in inflammatory bowel disease - eBioMedicine

Inflammatory bowel disease-associated ubiquitin ligase RNF183 promotes  lysosomal degradation of DR5 and TRAIL-induced caspase activation |  Scientific Reports
Inflammatory bowel disease-associated ubiquitin ligase RNF183 promotes lysosomal degradation of DR5 and TRAIL-induced caspase activation | Scientific Reports

A single early-in-life antibiotic course increases susceptibility to  DSS-induced colitis | Genome Medicine | Full Text
A single early-in-life antibiotic course increases susceptibility to DSS-induced colitis | Genome Medicine | Full Text

Plant green pigment of chlorophyllin attenuates inflammatory bowel diseases  by suppressing autophagy activation in mice | American Journal of  Physiology-Gastrointestinal and Liver Physiology
Plant green pigment of chlorophyllin attenuates inflammatory bowel diseases by suppressing autophagy activation in mice | American Journal of Physiology-Gastrointestinal and Liver Physiology

Peptidoglycan Recognition Proteins Protect Mice from Experimental Colitis by  Promoting Normal Gut Flora and Preventing Induction of Interferon-γ -  ScienceDirect
Peptidoglycan Recognition Proteins Protect Mice from Experimental Colitis by Promoting Normal Gut Flora and Preventing Induction of Interferon-γ - ScienceDirect

Frontiers | Glucocorticoid Therapy in Inflammatory Bowel Disease:  Mechanisms and Clinical Practice
Frontiers | Glucocorticoid Therapy in Inflammatory Bowel Disease: Mechanisms and Clinical Practice

Frontiers | The Molecular Mechanisms of Intestinal Inflammation and  Fibrosis in Crohn's Disease
Frontiers | The Molecular Mechanisms of Intestinal Inflammation and Fibrosis in Crohn's Disease

Leucrose, a natural sucrose isomer, suppresses dextran sulfate sodium  (DSS)-induced colitis in mice by regulating macrophage polarization via  JAK1/STAT6 signaling - ScienceDirect
Leucrose, a natural sucrose isomer, suppresses dextran sulfate sodium (DSS)-induced colitis in mice by regulating macrophage polarization via JAK1/STAT6 signaling - ScienceDirect

Biotin Supplementation Ameliorates Murine Colitis by Preventing NF-κB  Activation - Cellular and Molecular Gastroenterology and Hepatology
Biotin Supplementation Ameliorates Murine Colitis by Preventing NF-κB Activation - Cellular and Molecular Gastroenterology and Hepatology