Est vitamină Fatal markers of pulmonary fibrosis cast cuşcă sudic
IJMS | Free Full-Text | Idiopathic Pulmonary Fibrosis: Pathogenesis and the Emerging Role of Long Non-Coding RNAs | HTML
Sulforaphane prevents bleomycin‑induced pulmonary fibrosis in mice by inhibiting oxidative stress via nuclear factor erythroid 2‑related factor‑2 activation
Reprogramming of profibrotic macrophages for treatment of bleomycin‐induced pulmonary fibrosis | EMBO Molecular Medicine
Targeting Runt-Related Transcription Factor 1 Prevents Pulmonary Fibrosis and Reduces Expression of Severe Acute Respiratory Syndrome Coronavirus 2 Host Mediators - The American Journal of Pathology
IJMS | Free Full-Text | Idiopathic Pulmonary Fibrosis: Pathogenesis and the Emerging Role of Long Non-Coding RNAs | HTML
MBD2 serves as a viable target against pulmonary fibrosis by inhibiting macrophage M2 program | Science Advances
Frontiers | Dissecting the Role of Mesenchymal Stem Cells in Idiopathic Pulmonary Fibrosis: Cause or Solution
Fibroblasts and their responses to chronic injury in pulmonary fibrosis - ScienceDirect
The pathogenesis of pulmonary fibrosis: a moving target | European Respiratory Society
Differential Lysotracker Uptake Defines Two Populations of Distal Epithelial Cells in Idiopathic Pulmonary Fibrosis[v1] | Preprints
Vitamin D suppresses bleomycin-induced pulmonary fibrosis by targeting the local renin–angiotensin system in the lung | Scientific Reports
Idiopathic pulmonary fibrosis (IPF) signaling pathways and protective roles of melatonin - ScienceDirect
Idiopathic Pulmonary Fibrosis | NEJM
Idiopathic pulmonary fibrosis: Are any of the morphological-molecular markers useful in clinical management? | Semantic Scholar
Genetic Risk Factors for Idiopathic Pulmonary Fibrosis: Insights into | JIR
Molecular biomarkers in idiopathic pulmonary fibrosis | American Journal of Physiology-Lung Cellular and Molecular Physiology
Time for a change: is idiopathic pulmonary fibrosis still idiopathic and only fibrotic? - The Lancet Respiratory Medicine
Pulmonary fibrosis and COVID-19: the potential role for antifibrotic therapy - The Lancet Respiratory Medicine
The histone methyltransferase DOT1L is a new epigenetic regulator of pulmonary fibrosis | Cell Death & Disease